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The more data, the better, right? When it comes to genetics, it turns out that might not be the case.

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The Nintendo Switch exists, and is a fantastic gaming system that you can, in a pinch, play in a bar, a car, or on the train. Phones exist too, and the games on them.

If You Actually Want to Play Games, Get the New Nintendo 2. DS XLThe Nintendo Switch exists, and is a fantastic gaming system that you can, in a pinch, play in a bar, a car, or on the train. Phones exist too, and the games on them are better than ever. So why the hell should you own anything else? Because games. The Switch’s library is still small, and smartphones still lack those games you can get lost in for days. So if you want a mobile system that can go anywhere and play some of the best games ever designed, you need something from the Nintendo 3. DS family, which despite being seven years old, shows no signs of being at the end of its life any time soon.

What is it? A souped up 2. DS that finally folds. Like. It's a perfect blend of price and games. No Like. The hinge feels a little cheap. There’s have been six 3. DS consoles over the last seven years, each with some flaw that kept it from being the perfect blend of tech and affordability—but the New Nintendo 2. DS XL changes that.

As I write this review you’ll notice I capitalize “New” when talking about the New Nintendo 2. DS XL. That isn’t an affectation on my part. It’s part of Nintendo’s super confusing naming scheme. The New 2. DS, and the New 3.

DS launched back in 2. DS system. It’s not just a new chassis—these systems can play games the original 2. DS and 3. DS cannot, like Xenosaga Chronicles 3. D, or a wide range of SNES games including Donkey Kong Country, Street Fighter Alpha 2, and F- Zero. There aren’t that many exclusive games, but there are enough good ones that if you already own an older 3. DS system you’re probably wishing you had the new one. The New 2. DS, like the New 3.

DS, also has a faster processor, a micro. SD slot for on- console storage, NFC capabilities for all your Amiibos, two new buttons on the shoulders of the device, and a C- Stick analog joystick. This is, in every way, an upgrade over any non- New 2. DS, 3. DS or 3. DS XL you might own. The New Nintendo 2. DS XL isn’t just an update to the ugly old 2. DS that looked more like a bargain bin knock off than a system from the company that created mobile gaming.

With the new device, Nintendo ditches the 2. DS’s ugly slab design to mimic the folding design of every other 3. DS. At $1. 50, it’s $7.

DS and a full $6. New Nintendo 3. DS XL (the 3.

DS XL doesn’t include a charger, so I added that price to the total). This is now the mid- range mobile console, and unless you really, really like 3. D, it should be the one you buy.

Besides being cheaper than the New 3. DS XL, it’s also much lighter. It weighs 9. 2 ounces—the same as the original 2.

DS. The 3. DS XL weights 1. You might not consider that a major difference, but I definitely notice it when I toss the 3. DS into my purse, and I often forget I have the 2. DS in there. It’s also smaller, folded up, than both of the other consoles currently available in the 3.

DS lineup. The slab- like 2. DS is 5 inches by 5. The New 2. DS XL folds up to 3.

Which is not just significantly more pocketable than its predecessors, it’s also slightly smaller than the 3. DS XL, which folds up to 3. Unlike the original 2.

DS, this system isn’t punishing people for hating 3. D or wanting to save money. It’s the best 3. DS system available, only with a little size and one feature shaved off.

It even improves on the New 3. DS XL! That system has two big issues with it: The game cartridge is always visible, and always ugly, and you need a screwdriver and a little patience in order to replace the micro. SD card. The New 2. DS XL solves both problems by hiding both slots behind a little door on the bottom of the device.

But the 2. DS XL is not without a few minor problems. The speakers have been moved from the top half of the device to tiny slots on the bottom that your palms often cover. So when your palms cover the speakers the sound gets muffled, and when they’re uncovered the speakers sound far tinnier.

The sliders and hinge feel cheaper too, with the hinge having a little “give” to it (as illustrated to the left). The New 3. DS XL feels positively luxurious beside it. It’s the Lexus to this guy’s really solid Toyota Camry. As someone who has frequently driven a Camry, I am totally okay with that! The Nintendo 2. DS XL is more than enough machine for me. With the huge choice of fantastic DS, 3.

DS, and New 3. DS games on the system, and a brand new Metroid game just a month away, I don’t think this gadget’s gonna be leaving my bag any time soon. If you already own a New 3. DS XL, but never use the 3. D than this might be worth a look, and if you’re upgrading from the original 3. DS or just dipping your toes into legit mobile gaming, then this is the console to buy when it goes on sale July 2. READMEThe first 2.

DS that doesn’t force cheapskates and 3. D haters to compromise. It has a similar footprint to the 3. DS XL, but is much lighter. Feels a little more cheaply made than the 3.

DS XL, but still feels very durable. Plays a lot of really good games.

This Study is Forcing Scientists to Rethink the Human Genome. The more data, the better, right? When it comes to genetics, it turns out that might not be the case. As both genetic sequencing has gotten cheaper and computerized data analysis has gotten better, more and more researchers have turned to what are known as genome- wide association studies in hopes of sussing out which individual genes are associated with particular disorders. The logic here is simple: If you have a whole lot of people with a disease, you should be able to tell what genetic traits those people have in common that might be responsible.

This thinking has resulted in an entire catalogue of hundreds of research studies that has shed light on the genetic origins of diseases such as type 2 diabetes, Parkinson’s disease, Crohn’s disease, and prostate cancer, while helping fuel the rise of personalized medicine. Now, though, a new analysis calls the entire approach into question.

Writing in the journal Cell, a group of Stanford University geneticists write that such large studies are likely to produce genetic variants with little bearing on the disease in question—essentially false positives that confuse the results. Intuitively, one might expect disease- causing variants to cluster into key pathways that drive disease etiology [the causes of disease],” they write. But for complex traits, association signals tend to be spread across most of the genome—including near many genes without an obvious connection to disease.”Their analysis suggests an intriguing new way of viewing the genome in which nearly every gene impacts every other gene. Instead of a system in which you can plug and play different variables to affect different results, it’s a complex, inter- related network. They call this the “omnigenic model.”Their work has broad, sweeping implications for the entire field of genetics. First off, that all those big, expensive genome- wide association studies may wind up being little more than a waste of time because they turn up genetic variants that, while perhaps interconnected to the disease, may not actually point to a viable target for things like drug therapy. Indeed, genes that often seem related to diseases have stumped researchers in terms of the role they actually play in the condition.

In the paper, for example, the Stanford researchers re- analysed a 2. DNA variants linked to height—but only 1. In the paper, the Stanford researchers suggest that the impact of each variant has a teeny impact on height. Far from solving a problem though, this new research merely opens up an entirely new line of questioning—and shows us once again, that we may not know nearly as much as we thought we did.